This title appears in the Scientific Report :
2010
Please use the identifier:
http://dx.doi.org/10.1117/12.854430 in citations.
Localized plasmonic losses at metal back contacts of thin-film silicon solar cells
Localized plasmonic losses at metal back contacts of thin-film silicon solar cells
As liver fibrosis is the result of persistent necroinflammation in the liver, pro-inflammatory cytokines secreted in response to cell injury have a central role in the pathogenesis of liver fibrosis. We aimed to investigate the association of cytokine gene polymorphism and liver fibrosis among Chine...
Saved in:
Personal Name(s): | Paetzold, U. W. |
---|---|
Hallermann, F. / Pieters, B. E. / Rau, U. / Carius, R. / and von Plessen, G. | |
Contributing Institute: |
Photovoltaik; IEF-5 JARA-ENERGY; JARA-ENERGY |
Published in: | SPIE newsroom, 7725 (2010) S. 772517 |
Imprint: |
2010
|
Physical Description: |
772517 |
PubMed ID: |
17645476 |
DOI: |
10.1117/12.854430 |
Document Type: |
Journal Article |
Research Program: |
Erneuerbare Energien |
Series Title: |
SPIE Newsroom
7725 |
Subject (ZB): | |
Publikationsportal JuSER |
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700 | 1 | |0 P:(DE-HGF)0 |a Hallermann, F. |b 1 | |
700 | 1 | |0 P:(DE-Juel1)VDB86995 |a Pieters, B. E. |b 2 |u FZJ | |
700 | 1 | |0 P:(DE-Juel1)130285 |a Rau, U. |b 3 |u FZJ | |
700 | 1 | |0 P:(DE-Juel1)VDB4964 |a Carius, R. |b 4 |u FZJ | |
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520 | |a As liver fibrosis is the result of persistent necroinflammation in the liver, pro-inflammatory cytokines secreted in response to cell injury have a central role in the pathogenesis of liver fibrosis. We aimed to investigate the association of cytokine gene polymorphism and liver fibrosis among Chinese patients with chronic hepatitis B.Polymorphisms at interleukin-10 (IL-10-627, -1117), interleukin-1-beta (IL-1beta-511, -31, -3964), interleukin-1 receptor antagonist (IL-1RN), and tumor necrosis factor-alpha (TNF-alpha-308, -238) among Chinese chronic hepatitis B patients were determined. Severe liver fibrosis was defined as Ishak fibrosis score = 4 (of 6).Fifty-nine of 273 (22%) patients had severe fibrosis. The distribution of genotypes for IL-10-627 was CC (11%), CA (41%), and AA (48%). The CC genotype at IL-10-627 was protective against severe fibrosis (odds ratio (OR) 0.11; 95% CI 0.014-0.82; P = 0.032). After adjusted for baseline variables, the adjusted OR of CC genotypes at IL-10-627 for severe fibrosis was 0.063 (95% CI 0.06-0.64; P = 0.063). Other gene polymorphisms at IL-1beta, IL-1RN, TNF-alpha, and IL-10 had no significant association with severe fibrosis. Weak linkage disequilibrium was observed between IL-10-627 and IL-10-1117 with linkage disequilibrium coefficient of 0.12 (P < 0.001). The distribution of haplotypes of IL-10-1117 and IL-10-627 was A-A (69%), A-C (26%), and G-C (5%). High and intermediate IL-10 production (A-C and G-C) haplotypes were protective against severe fibrosis (OR 0.62; 95% CI 0.39-0.99; P = 0.046).High production genotype and haplotypes of IL-10 were associated with less severe liver fibrosis in chronic hepatitis B in Chinese. | ||
650 | 2 | |2 MeSH |a Adult | |
650 | 2 | |2 MeSH |a Female | |
650 | 2 | |2 MeSH |a Hepatitis B, Chronic: complications | |
650 | 2 | |2 MeSH |a Humans | |
650 | 2 | |2 MeSH |a Interleukin 1 Receptor Antagonist Protein: genetics | |
650 | 2 | |2 MeSH |a Interleukin-10: genetics | |
650 | 2 | |2 MeSH |a Interleukin-1beta: genetics | |
650 | 2 | |2 MeSH |a Liver Cirrhosis: complications | |
650 | 2 | |2 MeSH |a Liver Cirrhosis: genetics | |
650 | 2 | |2 MeSH |a Male | |
650 | 2 | |2 MeSH |a Polymorphism, Genetic | |
650 | 2 | |2 MeSH |a Tumor Necrosis Factor-alpha: genetics | |
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